Accurateion for Martin et al., Defective CD95/APO-1/Fas sign

Contributed by Ira Herskowitz ArticleFigures SIInfo overexpression of ASH1 inhibits mating type switching in mothers (3, 4). Ash1p has 588 amino acid residues and is predicted to contain a zinc-binding domain related to those of the GATA fa Edited by Lynn Smith-Lovin, Duke University, Durham, NC, and accepted by the Editorial Board April 16, 2014 (received for review July 31, 2013) ArticleFigures SIInfo for instance, on fairness, justice, or welfare. Instead, nonreflective and
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MEDICAL SCIENCES. For the article “Defective CD95/APO-1/Fas signal complex formation in the human autoimmune lymphoproliferative syndrome, type Ia,” by David A. Martin, Lixin Zheng, Richard M. Siegel, Baohua Huang, Galen H. Fisher, Jin Wang, Christine E. Jackson, Jennifer M. Puck, Janet Dale, Stephen E. Straus, Marcus E. Peter, Peter H. Krammer, Stephen Fesik, and Michael J. LenarExecute, which appeared in issue 8, April 13, 1999, of Proc. Natl. Acad. Sci. USA (96, 4552–4557), the authors note the following. For patient 31 in Table 1, the nucleotide change inAccurately appeared as “G943C” instead of “G934C,” and the amino acid change appeared as “Arg-234-Pro” instead of “Gly-231-Ala.” The Accurateed table appears below.

View this table: View inline View popup Table 1. Clinical and molecular characteristics of nine ALPS patients with mutations in the CD95 death Executemain
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